Fits, Faints & Funny Turns

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Crucial to establishing the correct diagnosis is a
detailed and accurate history preferably from both the patient and
eyewitnesses - unfortunately eye witnesses are not always reliable or
available and patient recollections may be sketchy!
The following points are suggestive of a cardiac aetiology
- loss of consciousness preceding convulsions, precipitation by exercise
or by being startled, family history of sudden and unexplained death,
family history of deafness in the patient or other family member, patients
whose convulsions respond to anticonvulsants but continue to have episodes
of loss of consciousness, some medications can cause arrhythmias. |
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A syncopal attack or faint is a sudden loss of
consciousness and postural tone associated with a drop in cerebral
blood flow, usually as a consequence of peripheral vascular reflex
mechanisms, and much more rarely as a consequence of primary cardiac
events. It can be difficult to distinguish faints from convulsions.
Faints are rare in a young child unless precipitated by a painful
stimulus (reflex anoxic seizures but common from early puberty to
late adolescence. A faint usually occurs on change of posture or on
prolonged standing (as in school assembly) whilst a convulsion may
occur at any time (particularly on awakening or going to sleep).
Loss of colour or limpness can occur with either a faint or
convulsion. Loss of consciousness is more gradual in a faint than in
a convulsion and is commonly associated with preceding pallor,
sweating and blurred vision. Convulsive movements can occur during a
faint. Severe and recurrent faints are often called
neuro-cardiogenic syncope (neurally-mediated syncope or
vasodepressor syncope) - profound bradycardia including asystole may
occur and may be accompanied by a range of clinical manifestations
including almost all the seizure types associated with epilepsy
although their duration is usually shorter than their epileptic
equivalents. |
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Syncopal attacks can occur due to arrhythmias such as
heart block (congenial or acquired), the sick-sinus syndrome, paroxysmal
tachycardia and the prolonged QT interval (with or without deafness) or to
structural cardiac disease e.g. severe aortic stenosis and pulmonary
stenosis, tetralogy of Fallot (spells) and cardiomyopathy – both
hypertrophic and dilated, primary and severe secondary pulmonary
hypertension.
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Investigations will be dependent upon the history and
examination but should include an ECG and echocardiogram. Those with
exercise induced symptoms should have an exercise test. A 24 Holter
monitor should be considered for those in whom an arrhythmia is strongly
suspected. Tilt testing may also be appropriate.
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This page was
last edited
16/2/2004 |